Scientists discover new mechanisms by which animal bodies suppress inflammation
February 26, 2018 Source: Xinhuanet
Window._bd_share_config={ "common":{ "bdSnsKey":{ },"bdText":"","bdMini":"2","bdMiniList":false,"bdPic":"","bdStyle":" 0","bdSize":"16"},"share":{ }};with(document)0[(getElementsByTagName('head')[0]||body).appendChild(createElement('script')) .src='http://bdimg.share.baidu.com/static/api/js/share.js?v=89860593.js?cdnversion='+~(-new Date()/36e5)];German scientists have discovered that the "oxygen monitoring system" in animals can promote the secretion of anti-inflammatory substances by immune B cells and inhibit the inflammatory response. It is a means of preventing the inflammation from exceeding the necessary range. The new findings will help develop new therapies for autoimmune diseases such as rheumatoid arthritis and colitis.
Inflammation is a defensive reaction to the body when it is traumatized or infected. It is usually beneficial, but it also needs to be terminated in time. If the immune system is too active, it can cause long-term inflammation and form an autoimmune disease by attacking its own tissue as a foreign object.
The animal's body closely monitors the oxygen level of the cells. Insufficient oxygen activates a class of proteins called hypoxia-inducible factor-1 and regulates a variety of physiological responses. Researchers at the University of Erlangen-Nuremberg in Germany have found that HIF-1α in these proteins plays an important role in inhibiting inflammation. In the absence of this protein, B cells cannot normally secrete anti-inflammatory substances, resulting in an exacerbation of autoimmune diseases such as arthritis or encephalomyelitis in experimental rats.
Related papers were published in the British journal Nature Communications.
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